Biomedcode's mouse model offers the in vivo validation of the efficacy of an improved tacrolimus...

Biomedcode coauthors a publication in the Journal of Controlled Release on the in vivo non-clinical proof of  the efficacy of a novel...

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Biomedcode is participating at the BTCure workshop on Animal models of Disease (18-20 Sept 2016) with talks...

BTCure is an IMI project co-funded by the European Union and the European Federation of Pharmaceutical Industries and Associations (EFPIA). The 4th...

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Biomedcode coauthors a review paper on aging related frailty in mice and tools used for its assessment

Dr N.Karagianni, Biomedcode's CSO, participates as an expert at MouseAGE COST action a European network for preclinical testing of interventions in...

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Biomedcode’s preclinical models: Priming drugs for success but also … predicting drug failure!

A new clinical study published by GlaxoSmithKline confirms findings in Biomedcode's TNF-DARE models.

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Review: animal models as a tool to dissect pivotal pathways driving spondyloarthritis

A new review highlights Biomedcode's models of spondyloarthropathy

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A recent review in the Nature Reviews Immunology highlights the instrumental role of the Tg197 model of...

Brenner et al., 2015. "Regulation of tumour necrosis factor signalling: live or let die". Nat Rev Immunol. May 26;15(6):362-74.

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Biomedcode's researchers co-authored a new paper that highlights the dominant role of subepithelial...

M. Roulis, C. Nikolaou, E. Kotsaki, E. Kaffe, N. Karagianni, V. Koliaraki, K. Salpea, J. Ragoussis, V. Aidinis, E. Martini, C. Becker, H.R....

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Prof. Dr. George Kollias, Biomedcode's President, was awarded 2014 Carol Nachman Prize for Rheumatology.

The Prize is one of Germany's highest medical honours and the most prestigious international award for research in Rheumatology.

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Evaluation of the effect of Src Kinase inhibitor

Biomedcode Hellas SA researchers presented a poster on the evaluation of the effect of Src Kinase inhibitor in arthritis

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TNF Signaling in Keratinocytes Triggers Interleukin-24-Dependent Psoriasis-like Skin Inflammation

A new keratinocyte-intrinsic mechanism linking TNFR1, NF-κB, ERK, IL-24, IL-22R1, and STAT3 signaling to disease initiation.

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